Max in WT synaptoneurosomes, suggesting that Src signaling could possibly be downregulated in KI synapses. Alternatively, our capacity to rescue SERT operate in KI midbrain synaptoneurosomes through the inhibition of FAK signifies elevated FAK signaling downstream in the Pro32Pro33 mutant, as verified by increased pFAK localization in five-HT synapses. Our https://walti421mzl3.blogchaat.com/profile